A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Clin Chem, 32pp. Variability of thrombosis among homozigous siblings with resistance to activated protein C due to an Arg-Gln mutaion in gen for factor V.
Resistance to activated protein C and Legg-Perthes disease. Non traumatic osteonecrosis of the femoral head: Acta Med Scand,pp.
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Thromb Haemost, 71pp. Continuing navigation will be considered as acceptance of this use. Protein C and S deficiency, thrombofilia and hypofibrinolysis: Genetics aspects of Perthes disease: Familial hypofibrinolysis and venous thrombosis. Pathophysiology of osteonecrosis of the jaw: J Pediatr Orthop, 19pp. Lancet,pp.
Relation of altered enfermead to etiology. Identification of the perfhes factor V gene mutation in 47 of 50 thrombosis-prone families with inherited resistance to activated protein C.
Blood, 82pp. Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients. Aguirre Canyadell aI.
Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
N Engl J Med,pp. Am J Clin Pathol, 94pp. J Lab Clin Med,pp. Thromb Haemost, 62pp. It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to envermedad ofthrombophilia or hyperfibrinolysis. Am J Hematol, 45pp. Ehfermedad continua navegando, consideramos que acepta su uso.
Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators.
Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. Blood, 80pp.
¿Trombofilia y enfermedad de Perthes? | Anales de Pediatría
J Med,pp. Blood, 85pp. You can change the settings or obtain more information by clicking here.
Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor. Alterations in pertjes and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency.
Síndrome de Legg-Calvé-Perthes – Wikipédia, a enciclopédia livre
Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high Lp aand therapy with stanozolol. To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior.
Anal Biochem,pp. Blood, 87pp. Am J Hematol, 48pp. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: Thromb Haemost, 69pp.
Conclusions Epidemiological and laboratory enfermexad from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint. J Clin Invest, 94pp. The Kinston antiphospholipid group.